Alumni
Sophie Huschke
M.Sc. Molecular Nutrition,
Raman spectroscopic characterization of the linkage- and interaction mechanism of nanoparticles and drugs with hepatic stellate cells ,
Key interests: drug delivery, pathophysiology of liver fibrosis
Raman spectroscopic characterization of the linkage- and interaction mechanism of nanoparticles and drugs with hepatic stellate cells ,
Key interests: drug delivery, pathophysiology of liver fibrosis
Wanling Foo
M.Ing. Bioengeneering,
Selective targeting of hepatocytes using multifunctional block copolymer micelles to restore critical cellular signaling functions ,
Key interests: drug delivery, nanopharmacology, material science
Selective targeting of hepatocytes using multifunctional block copolymer micelles to restore critical cellular signaling functions ,
Key interests: drug delivery, nanopharmacology, material science
Dr. med. Steffen Metzler
Physician,
… aims to understand micro RNA-induced metabolic activation on the course and outcome of sepsis ,
Key interests: microRNA, metabolism, intensive care medicine
… aims to understand micro RNA-induced metabolic activation on the course and outcome of sepsis ,
Key interests: microRNA, metabolism, intensive care medicine
Kay-Jovana Benecke
Medical student,
Targeted PKC-Inhibition to prevent and resolute excretory liver failure in experimental sepsis ,
Key interests: liver biology, cholestasis, protein kinase signaling
Targeted PKC-Inhibition to prevent and resolute excretory liver failure in experimental sepsis ,
Key interests: liver biology, cholestasis, protein kinase signaling
Micha Ploner
Medical student,
PKC-Isoforms as regulator of septic cholestasis in the PI3K/PKC pathway,
Key interests: liver biology, protein kinase signaling, membrane biology
PKC-Isoforms as regulator of septic cholestasis in the PI3K/PKC pathway,
Key interests: liver biology, protein kinase signaling, membrane biology
Michael Zschach, B.Sc.
untersucht die metabolische Wirtsantwort auf Infektionen.
Ling Xiong, M.Sc.
untersucht die Auswirkungen der Hemmung der Proteinkinase-C-Isoformen auf die Leberfunktion und immunmetabolische Folgen der Sepsis.